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Consistent with the diathesis-stress model, the two-hit hypothesis ( i.e ., early genetic and environmental discrepancies produce long-term vulnerability to a “second stress hit” that leads to the emergence of symptomatology) and its meta-evolved three-hit hypothesis ( i.e ., hit-1: genetic predisposition, hit-2: early-life environment, and hit-3: later-life environment) suggest that, in a given environmental context, vulnerability is enhanced and symptomatology emerges when failure to cope with adversity accumulates [169, 170] Celestia White Slim Garcinia Bundle – Ideal Beauty Deals KSA SHADE:HAZELNUT https://nypost.com/wp-content/uploads/sites/2/2024/03/face-ballooned-poisoned-body-holiday-78201252.jpg?quality=75&strip=all&w=744 2X

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